Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More

ADH1B Mouse anti-Human, Rat, Clone: OTI4F12, liquid, TrueMAB™ Non-distribution product as customer accommodation.

Mouse Monoclonal Antibody

Manufacturer:  OriGeneSupplier Diversity Partner TA502777

Catalog No. 50-167-3789


Add to cart

Description

Description

The protein encoded by this gene is a member of the alcohol dehydrogenase family. Members of this enzyme family metabolize a wide variety of substrates, including ethanol, retinol, other aliphatic alcohols, hydroxysteroids, and lipid peroxidation products. This encoded protein, consisting of several homo- and heterodimers of alpha, beta, and gamma subunits, exhibits high activity for ethanol oxidation and plays a major role in ethanol catabolism. Three genes encoding alpha, beta and gamma subunits are tandemly organized in a genomic segment as a gene cluster.
Specifications

Specifications

ADH1B
Monoclonal
0.89 mg/mL
PBS with 1% BSA, 50% glycerol and 0.02% sodium azide
P00325, Q66HF8
ADH2, HEL-S-117
Mouse
IgG2a
100 μL
-20° C, Avoid Freeze/Thaw Cycles
Primary
125, 298079
Flow Cytometry, Immunocytochemistry, Immunofluorescence, Immunohistochemistry (Paraffin), Western Blot
OTI4F12
Unconjugated
ADH1B
Liquid
ADH1B, Aldh1b1
Full length human recombit protein of human ADH1B produced in HEK293T cell.
Affinity Chromatography
RUO
Antibody
Monoclonal
Human, Rat
Documents
Provide Content Correction

The Fisher Scientific Encompass Program offers items which are not part of our distribution portfolio. These products typically do not have pictures or detailed descriptions. However, we are committed to improving your shopping experience. Please use the form below to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Cancel Submit