Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More

Alcohol Dehydrogenase 1B Mouse anti-Human, Clone: OTI3F2, Invitrogen™

Mouse Monoclonal Antibody

Manufacturer:  Invitrogen MA525547

Catalog No. PIMA525547


Add to cart

Description

Description

This gene encodes a member of the alcohol dehydrogenase family. The encoded protein is the alpha subunit of class I alcohol dehydrogenase, which consists of several homo- and heterodimers of alpha, beta and gamma subunits. Alcohol dehydrogenases catalyze the oxidation of alcohols to aldehydes. This gene is active in the liver in early fetal life but only weakly active in adult liver. This gene is found in a cluster with six additional alcohol dehydrogenase genes, including those encoding the beta and gamma subunits, on the long arm of chromosome 4. Mutations in this gene may contribute to variation in certain personality traits and substance dependence.
Specifications

Specifications

Alcohol Dehydrogenase 1B
Monoclonal
0.61 mg/mL
PBS with 1% BSA, 50% glycerol and 0.02% sodium azide; pH 7.3
P00325
ADH2, ADH, beta subunit, alcohol dehydrogenase 1B, alcohol dehydrogenase 2 (class I), beta polypeptide, alcohol dehydrogenase subunit beta, aldehyde reductase
Mouse
IgG1
100 μL
-20° C, Avoid Freeze/Thaw Cycles
Primary
125
Flow Cytometry, Immunohistochemistry (Paraffin), Western Blot
OTI3F2
Unconjugated
ADH1B
Liquid
ADH1B
Full length human recombit protein of ADH1B produced in HEK293T cell
Affinity Chromatography
RUO
Antibody
Monoclonal
Human
Documents
Provide Content Correction

We continue to work to improve your shopping experience and your feedback regarding this content is very important to us. Please use the form below to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Cancel Submit