Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More

Caveolin 1 Goat anti-Human, Polyclonal, Invitrogen™

Goat Polyclonal Antibody

Manufacturer:  Invitrogen PA19021

Catalog No. 01-674-605


Add to cart

Description

Description

This antibody is predicted to react with canine, mouse, porcine and rat based on sequence homology.

Caveolin-1 or CAV1 is a scaffolding proteins within caveolar membranes. It interacts directly with G-protein alpha subunits and can functionally regulate their activity. Caveolin-1 is also involved in the co-stimulatory signal essential for T-cell receptor (TCR) mediated T-cell activation. It can bind with DPP4 and induces T-cell proliferation and NF-kappa-B activation in a T-cell receptor/CD3-dependent manner. Mutations affecting the CAV1 gene can result in congenital generalized lipodystrophy 3, pulmonary hypertension primary 3, or partial lipodystrophy/congenital cataracts, and neurodegeneration syndrome.
Specifications

Specifications

Caveolin 1
Polyclonal
Unconjugated
CAV1
Liquid
CAV1
Internal sequence corresponding to amino acids C-DELSEKQVYDAH.
Antigen affinity chromatography
RUO
Antibody
Polyclonal
Human
Western Blot
0.5 mg/mL
TBS with 0.5% BSA and 0.02% sodium azide; pH 7.3
Q03135
CAV1; caveolin 1, caveolae protein, 22kDa ; HGNC:1527; CAV; MSTP085; VIP21 ; caveolae protein, 22-kD; caveolin 1; caveolin 1 caveolae protein, 22kD; c
Goat
IgG
100 μg
-20° C, Avoid Freeze/Thaw Cycles
Primary
857
Documents
Provide Content Correction

We continue to work to improve your shopping experience and your feedback regarding this content is very important to us. Please use the form below to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Cancel Submit