Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More

CD5/CD20 Mouse anti-Human, FITC, PE, Invitrogen™

Antibody Cocktail

Manufacturer:  Invitrogen MA112138

Catalog No. 01-670-857


Add to cart

Description

Description

This antibody is a monoclonal mixture of FITC-conjugated CD5 (clone M28623, isotype IgG2a) and PE-conjugated CD20 (clone DFA-7, isotype IgG1).

In humans, CD5 is a 67kDa T lymphocyte single chain transmembrane glycoprotein. It is present on all mature T lymphocytes, on most of thymocytes and on many T cell leukemias and lymphomas. It reacts with a subpopulation of activated B cells. The CD20 antigen is present on human pre-B lymphocytes and on B-lymphocytes atall stage of maturation, except on plasma cells. Low level expression of the CD20 antigen has been detected on normal T lymphocytes. CD20 is a non glycosylated protein with a molecular weight of 35 or 37 kDa depending on the degree of phosphorylation.
Specifications

Specifications

CD5/CD20
Cocktail
FITC, PE
MS4A1
Liquid
CD5, MS4A1
human T-acute lymphoblastic leukemia (ALL) cells and LB lymphoblastoid cell line
Protein A/G
RUO
Antibody
Monoclonal
Human
Flow Cytometry
DFA-7
PBS with 0.2% carrier protein and 0.08% sodium azide
P06127, P11836
CD5 antigen p56-62, T-cell surface glycoprotein CD5, lymphocyte antigen T1/Leu-1, B1, Bp35, CD20, LEU-16, MGC3969, MS4A1
Mouse
IgG1/IgG2a
100 Tests
4° C, do not freeze
Primary
921, 931
Documents
Provide Content Correction

We continue to work to improve your shopping experience and your feedback regarding this content is very important to us. Please use the form below to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Cancel Submit