Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More

ChREBP Rabbit anti-Human, Mouse, DyLight® 650, Polyclonal, Invitrogen™

Rabbit Polyclonal Antibody

Manufacturer:  Invitrogen PA522927

Catalog No. PIPA522927


Add to cart

Description

Description

The target sequence has 83% sequence homology with mouse and rat ChREBP.

ChREBP is a transcription factor involved in activating genes that encode enzymes of fatty acid biosynthesis in liver and adipose. This protein may have relevance to lipid metabolism, obesity, and type II diabetes.
Specifications

Specifications

CHREBP
Polyclonal
DyLight™ 650
MLXIPL
Liquid
MLXIPL
An internal synthetic peptide made to the human ChREBP protein sequence (between residues 50-150).
Antigen affinity chromatography
RUO
Antibody
Polyclonal
Human, Mouse
Immunocytochemistry, Immunofluorescence
1 mg/mL
sodium borate with no preservative
Q9NP71, Q99MZ3
CHREBP, MIO, MONDOB, WBSCR14, WS-bHLH, bHLHd14, MLX-interacting protein-like, Mlx interactor, WS basic-helix-loop-helix leucine zipper protein, Williams Beuren syndrome chromosome region 14, Williams-Beuren syndrome chromosome region 14 protein 1, Williams-Beuren syndrome chromosome region 14 protein 2, carbohydrate response element binding protein, carbohydrate-responsive element-binding protein, class D basic helix-loop-helix protein 14, williams-Beuren syndrome chromosomal region 14 protein
Rabbit
IgG
100 μL
4° C, store in dark
Primary
51085, 58805
Documents
Provide Content Correction

We continue to work to improve your shopping experience and your feedback regarding this content is very important to us. Please use the form below to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Cancel Submit