Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More

MKP2 Rabbit anti-Human, Mouse, Polyclonal, Invitrogen™

Rabbit Polyclonal Antibody

Manufacturer:  Invitrogen PA515549

Catalog No. PIPA515549


Add to cart

Description

Description

DUSP4 is a member of the dual specificity protein phosphatase subfamily. These phosphatases inactivate their target kinases by dephosphorylating both the phosphoserine/threonine and phosphotyrosine residues. They negatively regulate members of the mitogen-activated protein (MAP) kinase superfamily (MAPK/ERK, SAPK/JNK, p38), which are associated with cellular proliferation and differentiation. Different members of the family of dual specificity phosphatases show distinct substrate specificities for various MAP kinases, different tissue distribution and subcellular localization, and different modes of inducibility of their expression by extracellular stimuli. DUSP4 inactivates ERK1, ERK2 and JNK, is expressed in a variety of tissues, and is localized in the nucleus.
Specifications

Specifications

MKP2
Polyclonal
PBS with 0.09% sodium azide
Q13115, Q8BFV3
Mitogen-activated protein kinase phosphatase 2; MAP kinase phosphatase 2; MKP-2; Dual specificity protein phosphatase hVH2;
Rabbit
IgG
400 μL
-20° C, Avoid Freeze/Thaw Cycles
Primary
1846, 319520
Immunohistochemistry (Paraffin), Western Blot
Unconjugated
DUSP4
Liquid
DUSP4
KLH conjugated synthetic peptide between 331-361 amino acids from the C-terminal region of human DUSP4
Ammonium sulfate precipitation, Size-exclusion - Dialysis
RUO
Antibody
Polyclonal
Human, Mouse
Documents
Provide Content Correction

We continue to work to improve your shopping experience and your feedback regarding this content is very important to us. Please use the form below to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Cancel Submit