Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More

FTO Rabbit anti-Human, Mouse, Non-human primate, Polyclonal, Invitrogen™

Rabbit Polyclonal Antibody

Manufacturer:  Invitrogen PA146310

Catalog No. PA146310


Add to cart

Description

Description

Suggested positive control: human brain lysate, human brain protein.

FTO is a novel protein and a member of the non-heme dioxygenase (Fe(II)-and 2-oxoglutarate-dependent dioxygenases) superfamily. Amino acid conservation patterns support this hypothesis and indicates that both 2-oxoglutarate and iron should be important for FTO function. Though not much is yet known about FTO, it is thought that the protein may somehow modify the activity of genes involved in metabolism and fat storage, which in turn may influence a person ft.s risk of obesity.
Specifications

Specifications

FTO
Polyclonal
Unconjugated
FTO
Liquid
FTO
Synthetic peptide corresponding to an C-terminal portion within residues 400-505 of the human protein. [Swiss-ProtNo. Q9C0B1]
Antigen affinity chromatography
RUO
Antibody
Polyclonal
Human, Mouse, Non-human primate
Western Blot
1.1 mg/mL
tris citrate, tris phosphate with 0.1% sodium azide; pH 7-8
Q9C0B1, Q8BGW1
fat mass and obesity associated protein , fatso , MGC5149 , KIAA1752
Rabbit
IgG
100 μL
Store at 4°C short term. For long term storage, store at -20°C, avoiding freeze/thaw cycles.
Primary
26383, 79068
Documents
Provide Content Correction

We continue to work to improve your shopping experience and your feedback regarding this content is very important to us. Please use the form below to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Cancel Submit