Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More

GCNT2 Rabbit anti-Human, Mouse, Polyclonal, Invitrogen™

Rabbit Polyclonal Antibody

Manufacturer:  Invitrogen PA523721

Catalog No. PIPA523721


Add to cart

Description

Description

This gene encodes the enzyme responsible for formation of the blood group I antigen. The i and I antigens are distinguished by linear and branched poly-N-acetyllactosaminoglycans, respectively. The encoded protein is the I-branching enzyme, a beta-1,6-N-acetylglucosaminyltransferase responsible for the conversion of fetal i antigen to adult I antigen in erythrocytes during embryonic development. Mutations in this gene have been associated with adult i blood group phenotype. Alternatively spliced transcript variants encoding different isoforms have been described.
Specifications

Specifications

GCNT2
Polyclonal
PBS with 0.09% sodium azide
Q8N0V5, P97402
CCAT, GCNT2C, GCNT5, IGNT, II, NACGT1, NAGCT1, ULG3, bA360O19.2, bA421M1.1, I beta-1,6-N-acetylglucosaminyltransferase, Ii blood group, N-acetyllactosaminide beta-1,6-N-acetylglucosaminyl-transferase, beta-1,6-N-acetylglucosaminyltransferase 2
Rabbit
IgG
400 μL
-20° C, Avoid Freeze/Thaw Cycles
Primary
14538, 2651
Immunohistochemistry (Paraffin), Western Blot
Unconjugated
GCNT2
Liquid
GCNT2
KLH conjugated synthetic peptide between 364-391 amino acids from the C-terminal region of human GCNT2
Protein A, Antigen Affinity Chromatography
RUO
Antibody
Polyclonal
Human, Mouse
Documents
Provide Content Correction

We continue to work to improve your shopping experience and your feedback regarding this content is very important to us. Please use the form below to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Cancel Submit