Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More

HIF-2 alpha Rabbit anti-Fish, Hamster, Human, Mouse, Non-human primate, Ovine, Rat, Biotin, Polyclonal, Invitrogen™

Rabbit Polyclonal Antibody

Manufacturer:  Invitrogen PA522693

Catalog No. PIPA522693

Add to cart



Suggested positive control: hypoxic lysates.

HIF-2 alpha (Epas1) is a transcription factor involved in the induction of genes regulated by oxygen. HIF-2 alpha contains a basic-helix-loop-helix domain protein dimerization domain as well as a domain found in proteins in signal transduction pathways which respond to oxygen levels. It also regulates the vascular endothelial growth factor (VEGF) expression and seems to be implicated in the development of blood vessels and the tubular system of the lungs. Mutations in this gene are associated with erythrocytosis familial type 4.


HIF-2 alpha
A peptide derived from the C-terminus of mouse/human HIF-2 alpha protein.
Antigen affinity chromatography
Fish, Hamster, Human, Mouse, Non-human primate, Ovine, Rat
Immunohistochemistry (Frozen), Immunohistochemistry (Paraffin), Western Blot
2 mg/mL
PBS with no preservative
Q99814, P97481, Q9JHS1
ECYT4, HIF2A, HLF, MOP2, PASD2, bHLHe73, EPAS-1, HIF-1-alpha-like factor, HIF-1alpha-like factor, HIF-2-alpha, HIF2-alpha, PAS domain-containing protein 2, basic-helix-loop-helix-PAS protein MOP2, class E basic helix-loop-helix protein 73, endothelial PAS domain-containing protein 1, hypoxia-inducible factor 2 alpha, hypoxia-inducible factor 2-alpha, member of PAS protein 2
100 μL
4° C, store in dark
100771160, 13819, 2034, 29452, 443286
Provide Content Correction

We continue to work to improve your shopping experience and your feedback regarding this content is very important to us. Please use the form below to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Cancel Submit