Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More

CD104 Mouse anti-Human, Alexa Fluor® 488, Clone: 422325, Invitrogen™

Mouse Monoclonal Antibody

Manufacturer:  Invitrogen MA523641

Catalog No. PIMA523641


Add to cart

Description

Description

In Western blots, no cross-reactivity with recombit human Integrin beta 1, beta 2, beta 3, beta 5, beta 6, beta 8, or recombit mouse Integrin beta 4 is observed.

Integrins are heterodimers comprised of alpha and beta subunits, that are noncovalently associated transmembrane glycoprotein receptors. Different combinations of alpha and beta polypeptides form complexes that vary in their ligand-binding specificities. Integrins mediate cell-matrix or cell-cell adhesion, and transduced signals that regulate gene expression and cell growth. This gene encodes the integrin beta 4 subunit, a receptor for the laminins. This subunit tends to associate with alpha 6 subunit and is likely to play a pivotal role in the biology of invasive carcinoma. Mutations in this gene are associated with epidermolysis bullosa with pyloric atresia. Multiple alternatively spliced transcript variants encoding distinct isoforms have been found for this gene.
Specifications

Specifications

CD104
Monoclonal
Alexa Fluor™ 488
ITGB4
Liquid
ITGB4
CHO-derived recombit human Integrin beta 4/CD104 Asn28-Ser710
Protein A/G
RUO
Antibody
Monoclonal
Human
Flow Cytometry
422325
Buffered saline solution with BSA and 0.09% sodium azide
P16144
CD104 antigen, GP150, integrin beta-4, integrin beta-4 subunit, ITGB4
Mouse
IgG2B
100 Tests
4° C, store in dark, DO NOT FREEZE!
Primary
3691
Documents
Provide Content Correction

We continue to work to improve your shopping experience and your feedback regarding this content is very important to us. Please use the form below to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Cancel Submit