Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More

KCNT2 Rabbit anti-Human, Polyclonal, Invitrogen™

Rabbit Polyclonal Antibody

Manufacturer:  Invitrogen PA523855

Catalog No. PIPA523855


Add to cart

Description

Description

Kcnt2 calcium (Ca2+) -activated potassium channels (KCa) are a group of 6/7-TM ion channels that selectively transport K+ ions across biological membranes. KCa channels are broadly classified into three subtypes: SK, IK and BK channels (small, intermediate and big conductance). Kcnt2 is an outward rectifying potassium channel, and produces rapidly activating outward rectifier K(+) currents. Kcnt2 is activated by high intracellular sodium and chloride levels. Further, Kcnt2 channel activity is inhibited by ATP and by inhalation anesthetics, such as isoflurane. Functionally, Kcnt2 is inhibited upon stimulation of G-protein coupled receptors, such as CHRM1 and GRIA1.
Specifications

Specifications

KCNT2
Polyclonal
PBS with 0.09% sodium azide
Q6UVM3
KCa4.2, SLICK, SLO2.1, potassium channel subfamily T member 2, sequence like an intermediate conductance potassium channel subunit, sodium activated potassium channel, sodium and chloride activated ATP sensitive potassium channel, sodium and chloride-activated ATP-sensitive potassi
Rabbit
IgG
400 μL
-20° C, Avoid Freeze/Thaw Cycles
Primary
343450
Flow Cytometry, Immunohistochemistry (Paraffin), Western Blot
Unconjugated
KCNT2
Liquid
KCNT2
KLH conjugated synthetic peptide between 1069-1097 amino acids from the C-terminal region of human KCNT2
Antigen Affinity Chromatography, Protein A
RUO
Antibody
Polyclonal
Human
Documents
Provide Content Correction

We continue to work to improve your shopping experience and your feedback regarding this content is very important to us. Please use the form below to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Cancel Submit