Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More

Phenobarbital Mouse anti-Chemical, Clone: PB9-30.17.4.16, Invitrogen™

Mouse Monoclonal Antibody

Manufacturer:  Invitrogen MA110741

Catalog No. 01-671-971


Add to cart

Description

Description

MA1-10741 detects Phenobarbital. MA1-10741 has been successfully used in ELISA applications. The MA1-10741 immunogen is Phenobarbital, conjugated to KLH.

Phenobarbital is a long-acting barbituric acid derivative with antipsychotic property. Phenobarbital binds to and activates the gamma-aminobutyric acid (GABA)-A receptor, thereby mimicking the inhibitory actions of GABA in the brain. The activation effects of the phenobarbital-receptor-ionophore complex include increased frequency of chloride channel openings, membrane hyperpolarization and ultimately synaptic inhibition and decreased neuronal excitability. In addition, this agent inhibits glutamate induced depolarization. [National Center for Biotechnology Information. PubChem Compound Database; CID=4763]
Specifications

Specifications

Phenobarbital
Monoclonal
1.8 mg/mL
PBS with no preservative; pH 7.4
Luminal; Phenobarbitone; Phenobarbitol; Phenylethylbarbiturate; Phenobarbituric acid; C12H12N2O3
Phenobarbital, conjugated to KLH.
Purified
RUO
Antibody
Monoclonal
ELISA
PB9-30.17.4.16
Unconjugated
Liquid
Mouse
IgG1
500 μg
-20° C, Avoid Freeze/Thaw Cycles
Primary
Chemical
Documents
Provide Content Correction

We continue to work to improve your shopping experience and your feedback regarding this content is very important to us. Please use the form below to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Cancel Submit