Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More

PIK3C2B Rabbit anti-Human, Polyclonal, Invitrogen™

Rabbit Polyclonal Antibody

Manufacturer:  Invitrogen PA515238

Catalog No. PIPA515238

This item has been discontinued by the manufacturer and is no longer available. Please call customer service for assistance: 1-800-766-7000.


Description

Description

Protein kinases are enzymes that transfer a phosphate group from a phosphate donor, generally the g phosphate of ATP, onto an acceptor amino acid in a substrate protein. By this basic mechanism, protein kinases mediate most of the signal transduction in eukaryotic cells, regulating cellular metabolism, transcription, cell cycle progression, cytoskeletal rearrangement and cell movement, apoptosis, and differentiation. With more than 500 gene products, the protein kinase family is one of the largest families of proteins in eukaryotes. The family has been classified in 8 major groups based on sequence comparison of their tyrosine (PTK) or serine/threonine (STK) kinase catalytic domains.
Specifications

Specifications

PIK3C2B
Polyclonal
PBS with 0.09% sodium azide
O00750
Phosphatidylinositol-4-phosphate 3-kinase C2 domain-containing beta polypeptide, Phosphoinositide 3-Kinase-C2-beta, PtdIns-3-kinase C2 beta
Rabbit
IgG
400 μL
-20° C, Avoid Freeze/Thaw Cycles
Primary
5287
Immunohistochemistry
Unconjugated
PIK3C2B
Liquid
PIK3C2B
KLH conjugated synthetic peptide between 120-150 amino acids from the N-terminal region of human PI3KC2B
Ammonium sulfate precipitation, Size-exclusion - Dialysis
RUO
Antibody
Polyclonal
Human
Documents
Provide Content Correction

We continue to work to improve your shopping experience and your feedback regarding this content is very important to us. Please use the form below to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Cancel Submit