Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More

PLCB1 Rabbit anti-Human, Polyclonal, Invitrogen™

Rabbit Polyclonal Antibody

Manufacturer:  Invitrogen PA526381

Catalog No. PIPA526381

This item has been discontinued by the manufacturer and is no longer available. Please call customer service for assistance: 1-800-766-7000.


Description

Description

The protein encoded by this gene catalyzes the formation of inositol 1,4,5-trisphosphate and diacylglycerol from phosphatidylinositol 4,5-bisphosphate. This reaction uses calcium as a cofactor and plays an important role in the intracellular transduction of many extracellular signals. This gene is activated by two G-protein alpha subunits, alpha-q and alpha-11.
Specifications

Specifications

PLCB1
Polyclonal
PBS with 0.09% sodium azide
Q9NQ66
EIEE12, PI-PLC, PLC-154, PLC-I, PLC154, PLCB1A, PLCB1B, 1-phosphatidyl-D-myo-inositol-4,5-bisphosphate, 1-phosphatidylinositol-4,5-bisphosphate phosphodiesterase beta 1, 1-phosphatidylinositol-4,5-bisphosphate phosphodiesterase beta-1, PLC-beta-1, inositoltrisphosphohydrolase, monophosphatidylinositol phosphodieste
Rabbit
IgG
400 μL
-20° C, Avoid Freeze/Thaw Cycles
Primary
23236
Flow Cytometry, Western Blot
Unconjugated
PLCB1
Liquid
PLCB1
KLH conjugated synthetic peptide between 1148-1176 amino acids from the C-terminal region of human PLCB1
Ammonium sulfate precipitation, Size-exclusion - Dialysis
RUO
Antibody
Polyclonal
Human
Documents
Provide Content Correction

We continue to work to improve your shopping experience and your feedback regarding this content is very important to us. Please use the form below to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Cancel Submit