Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More

C13orf15 Mouse anti-Human, Clone: 3B9, Invitrogen™

Mouse Monoclonal Antibody

Manufacturer:  Invitrogen MA524460

Catalog No. PIMA524460


Add to cart

Description

Description

Peptide Sequence: MKPPAEDLSD ALCEFDAVLA DFASPFHERH FHYEEHLERM KRRSSASVSD SSGFSDSESA DSLYRNSFSF SDEKLNSPTD STPALLSATV TPQKAKLGDT KELEAFIADL DKTLASM

This gene is thought to regulate cell cycle progression. It is induced by p53 in response to DNA damage, or by sublytic levels of complement system proteins that result in activation of the cell cycle. The encoded protein localizes to the cytoplasm during interphase and to centrosomes during mitosis. The protein forms a complex with polo-like kinase 1. The protein also translocates to the nucleus in response to treatment with complement system proteins, and can associate with and increase the kinase activity of cell division cycle 2 protein. In different assays and cell types, overexpression of this protein has been shown to activate or suppress cell cycle progression.
Specifications

Specifications

C13orf15
Monoclonal
Unconjugated
RGCC
Liquid
RGCC
C13orf15 (NP_054778.1, 1 a.a. ∽ 117 a.a) partial recombit protein with GST tag. MW of the GST tag alone is 26 KDa.
Protein A
RUO
Antibody
Monoclonal
Human
ELISA, Immunocytochemistry, Immunofluorescence
3B9
PBS with no preservative; pH 7.4
Q9H4X1
RGC-32, RGC32, bA157L14.2, response gene to complement 32 protein
Mouse
IgG1, kappa
100 μg
-20° C
Primary
28984
Documents
Provide Content Correction

We continue to work to improve your shopping experience and your feedback regarding this content is very important to us. Please use the form below to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Cancel Submit