Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More

LAMP2 Mouse anti-Human, FITC, Clone: H4B4, Invitrogen™ Non-distribution product as customer accommodation.

Mouse Monoclonal Antibody

Manufacturer:  Invitrogen A15463

Catalog No. 50-113-7061


Add to cart

Description

Description

Lysosomes are membrane-bound vacuoles which play a critical role in cellular metabolism. These organelles participate in endocytosis, the synthesis and assembly of acid hydrolases, act as sites for digestion of foreign materials and for specialized autolytic cellular processes. Membrane-associated glycoproteins have an important role in normal lysosomal function. Prior to posttranslational modification, Lysosome Associated Membrane Protein 2 (LAMP 2) is an ~45 kDa polypeptide. Mature, functional LAMP 2 is extensively glycosylated with a variety of different N-linked and O-linked oligosaccharides with a total molecular weight of ~100-110 kDa. LAMP 2 has a structure comprised of a large amino-terminal intra-lysosomal domain, a hydrophobic trans-membrane domain, and a short carboxyl-terminal cytoplasmic tail.
Specifications

Specifications

LAMP2, CD107b
Monoclonal
0.1 mg/mL
PBS with 0.1% sodium azide
P13473
CD107b, LAMP-2, LAMPB, LGP110, CD107 antigen-like family member B, lysosome-associated membrane glycoprotein 2, lysosome-associated membrane protein 2
Mouse
IgG1
100 μg
Immunology
Antibody
Monoclonal
Human
Flow Cytometry
H4B4
FITC
LAMP2
Liquid
LAMP2
Human CD107b
Purified
RUO
4° C
Primary
3920
Documents
Provide Content Correction

The Fisher Scientific Encompass Program offers items which are not part of our distribution portfolio. These products typically do not have pictures or detailed descriptions. However, we are committed to improving your shopping experience. Please use the form below to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Cancel Submit