Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More

HIF-2 alpha Mouse anti-Human, Mouse, Rat, Clone: GT125, Invitrogen™

Mouse Monoclonal Antibody

Manufacturer:  Invitrogen MA527743

Catalog No. PIMA527743


Add to cart

Description

Description

Positive Control: HepG2 (1% O2 treatment for 24hr) Predicted Reactivity: Pig (83%) Store product as a concentrated solution. Centrifuge briefly prior to opening the vial. For short-term storage (1-2 weeks), product can be stored at 4°C. For long-term storage, aliquot and store product at -20° C or below, avioiding multiple freeze-thaw cycles.

HIF2 alpha encodes a transcription factor involved in the induction of genes regulated by oxygen, which is induced as oxygen levels fall. The encoded protein contains a basic-helix-loop-helix domain protein dimerization domain as well as a domain found in proteins in signal transduction pathways which respond to oxygen levels. Mutations in this gene are associated with erythrocytosis familial type 4.
Specifications

Specifications

HIF-2 alpha
Monoclonal
3 mg/mL
PBS with no preservative
P97481, Q99814, Q9JHS1
ECYT4, PASD2, bHLHe73, endothelial PAS domain protein 1, HIF2A, HLF, EPAS1, MOP2
Mouse
IgG1
100 μl
Store at 4°C short term. For long term storage, store at -20°C, avoiding freeze/thaw cycles.
Primary
13819, 2034, 29452
Immunocytochemistry, Immunofluorescence, Immunoprecipitation, Western Blot
GT125
Unconjugated
EPAS1
Liquid
EPAS1
Recombit protein encompassing a sequence within the center region of human HIF2 alpha. The exact sequence is proprietary.
Protein G
RUO
Antibody
Monoclonal
Human, Mouse, Rat
Documents
Provide Content Correction

We continue to work to improve your shopping experience and your feedback regarding this content is very important to us. Please use the form below to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Cancel Submit