Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More

Cdk4 Ab-1 Mouse Monoclonal Antibody, Epredia™

Ensure accurate and reproducible results in western blotting, immunofluorescence, immunoprecipitation and kinase assay applications.

Supplier:  Epredia MS299P1

 View more versions of this product

Catalog No. MS299P1


Add to Cart

Description

Description

A family of proteins designated as cdks are critical regulators of cell cycle progression. The prototype member of this family, p34cdc2, and a related protein cdk2, function late in the cycle while cdk4 and cdk6 are critically involved in G1 to S progression.

Host Species: Mouse

Clone: DCS-35

Isotype: IgG1/κ

Species Reactivity: Human, Mouse, Rat and Pig. Others not known.

Epitope: aa 1-20

Immunogen: Purified recombinant Cdk4 protein

Molecular Weight: 34 kDa

Positive Control: NIH 3T3, MAD109, LS174T, or MCF-7 cells

Cellular Localization: Nuclear and cytoplasmic

Recommended for:

  • Western Blotting
  • Immunofluorescence
  • Immunoprecipitation (Native only)
  • Kinase Assay

Note:

USA: RUO; Int'l: RUO

Specifications

Specifications

Cdk4 Ab-1
Monoclonal
200μg/mL
Mouse
500 μL
Cell Cycle
Human, Mouse, Porcine, Rat
IgG2a κ
Immunofluorescence, Immunoprecipitation, Western Blot
DCS-35
Unconjugated
Purified recombinant cdk4 protein
RUO
Primary
Concentrated
Product Suggestions

Product Suggestions

SDS
Documents

Documents

Product Certifications
Promotions

Promotions

Provide Content Correction

We continue to work to improve your shopping experience and your feedback regarding this content is very important to us. Please use the form below to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Cancel Submit