Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More

MYL7 Mouse anti-Human, Clone: OTI7F2, Invitrogen™

Mouse Monoclonal Antibody

Manufacturer:  Invitrogen MA526389

Catalog No. PIMA526389


Add to cart

Description

Description

Encoded by the MYL7 gene, myosin regulatory light chain 7, also designated Myosin regulatory light chain 2, atrial isoform (MLC-2a), is part of a hexameric complex of two heavy chains and four light chains predomitly expressed in adult atrial muscle. Myosin regulatory light chain 7 binds calcium and has been shown to be a useful molecular marker for cardiac chamber specification. The co-expression of myosin regulatory light chain 7 and myosin light chain 2 (MLC2v) in the outflow tract and atrioventricular canal, together with the single expression in the atrial (MYL7) and ventricular (MYL2) myo-cardium, permits the delineation of their boundaries. At the amino acid level there is 95% homology between the human and mouse myosin regulatory light chain 7 sequences.
Specifications

Specifications

MYL7
Monoclonal
1 mg/mL
PBS with 1% BSA, 50% glycerol and 0.02% sodium azide; pH 7.3
Q01449
MYL2A, MYLC2A, MLC-2a, MLC2a, myosin light chain 2a, myosin regulatory light chain 2, atrial isoform, myosin regulatory light chain 7, myosin, light polypeptide 7, regulatory
Mouse
IgG1
100 μL
-20° C, Avoid Freeze/Thaw Cycles
Primary
58498
Flow Cytometry, Immunohistochemistry (Paraffin), Western Blot
OTI7F2
Unconjugated
MYL7
Liquid
MYL7
Full length human recombit protein of MYL7 produced in E.coli
Affinity Chromatography
RUO
Antibody
Monoclonal
Human
Documents
Provide Content Correction

We continue to work to improve your shopping experience and your feedback regarding this content is very important to us. Please use the form below to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Cancel Submit