Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More

NNMT Mouse anti-Human, Mouse, Clone: OTI3D8, Invitrogen™

Mouse Monoclonal Antibody

Manufacturer:  Invitrogen MA525476

Catalog No. PIMA525476


Add to cart

Description

Description

Nicotinamide N-methyltransferase (NNMT) catalyzes the N-methylation of nicotin-amide and other pyridines. NNMT activity in the human liver has a bimodal frequency distribution, indicating that its enzyme activity may be modulated through a genetic polymorphism, which could have functional implications for individual differences in drug and xenobiotic toxicity. The gene that encodes human NNMT is approximately 16.5 kb in length, consists of three exons and two introns and maps to 11q23.2. NNMT isolated from the human liver was determined to be 969 nucleotides in length, with a 792 nucleotide open reading frame that encodes a 264 amino acid protein. The NNMT gene is presumed to be a significant genetic determit of plasma homocysteine levels in Spanish families, since it encodes an enzyme involved in homocysteine synthesis.
Specifications

Specifications

NNMT
Monoclonal
1 mg/mL
PBS with 1% BSA, 50% glycerol and 0.02% sodium azide; pH 7.3
P40261, O55239
Nicotinamide N-methyltransferase; NNMT
Mouse
IgG2b
100 μL
-20° C, Avoid Freeze/Thaw Cycles
Primary
18113, 4837
Flow Cytometry, Immunocytochemistry, Immunofluorescence, Immunohistochemistry (Paraffin), Western Blot
OTI3D8
Unconjugated
NNMT
Liquid
NNMT
Full length human recombit protein of NNMT produced in HEK293T cell
Affinity Chromatography
RUO
Antibody
Monoclonal
Human, Mouse
Documents
Provide Content Correction

We continue to work to improve your shopping experience and your feedback regarding this content is very important to us. Please use the form below to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Cancel Submit