Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More

Invitrogen™ OXSR1 Polyclonal Antibody

Rabbit Polyclonal Antibody

Supplier:  Invitrogen™ PA517297

Catalog No. PIPA517297


Only null left
Add to Cart

Description

Description

It is not recommended to aliquot this antibody. This antibody is not cross-reactive with SPAK or other members of the GCK family.

OXSR1 serine/threonine kinase or (OSR1) is activated by osmotic stresses and regulates downstream protein kinases such as PAK1 in order to establish the sensor/signaling modules that initiate the cellular response to environmental stress. OXSR1 can be phosphorylated by ATM or ATR during the DNA damage process. It regulates downstream kinases in response to environmental stress, and may play a role in regulating the actin cytoskeleton.
TRUSTED_SUSTAINABILITY
Specifications

Specifications

OXSR1
Polyclonal
0.01M HEPES with 100μg/mL BSA, 50% glycerol, 0.15M NaCl and no preservative; pH 7.5
O95747
Oxsr1
Synthetic peptide corresponding to residues at the amino terminus of human OSR1.
100 μL
Primary
Human, Monkey, Bovine
Antibody
IgG
Flow Cytometry, Western Blot, Immunocytochemistry
Unconjugated
Oxsr1
2210022N24Rik; 2810422B09Rik; AI462649; AW209236; C6orf182; CEP57R; KIAA1101; mKIAA1101; Osr1; OTTHUMP00000209062; oxidative stress responsive 1; oxidative stress responsive kinase 1; oxidative stress-responsive 1 protein; oxidative-stress responsive 1; OXSR1; Serine/threonine-protein kinase OSR1
Rabbit
Antigen Affinity Chromatography
RUO
526949, 9943
-20°C
Liquid
Product Suggestions

Product Suggestions

Videos
SDS
Documents

Documents

Product Certifications
Promotions

Promotions

Product Content Correction

Your input is important to us. Please complete this form to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Your feedback has been submitted: Thank you for helping us improve our website.