Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More

R&D Systems™ Mouse TIM-1/KIM-1/HAVCR Recombinant Protein

Manufacturer:  R&D Systems™ 1817TM050/CF

 View more versions of this product

Catalog No. 1817TM050CF


Add to cart
This item is not returnable. View return policy

Description

Description

Measured by its ability to induce IL-6 or TNF-alpha secretion by RAW 264.7 mouse monocyte/macrophage cells. The ED 50 for this effect is 0.1-0.4 µg/mL. Measured by its ability to bind THP-1 human acute monocytic leukemia cells. As determined by flow cytometric analysis, there is a greater than 3 fold increase in the fluorescence of apoptotic THP-1 cells treated with 0.5 µg/mL recombinant mouse TIM-1/His andMouse Anti-polyHistidine PE-conjugated Monoclonal Antibody (Catalog # IC050P ) compared to cells stained with anti-Mouse Anti-polyHistidine PE-conjugated Monoclonal Antibody alone.
Specifications

Specifications

Lyophilized from a 0.2μm filtered solution in Tris-Citrate and NaCl.
21.8 kDa
95%, by SDS-PAGE under reducing conditions and visualized by silver stain.
Mouse
Unlabeled
171283
TIM-1/KIM-1/HAVCR
50μg
Use a manual defrost freezer and avoid repeated freeze-thaw cycles. 12 months from date of receipt, -20 to -70° C as supplied. 1 month, 2 to 8° C under sterile conditions after reconstitution. 3 months, -20 to -70° C under sterile conditions after reconstitution.
Yes
Documents
Provide Content Correction

We continue to work to improve your shopping experience and your feedback regarding this content is very important to us. Please use the form below to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Cancel Submit