Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More
Our pricing system is unavailable. You are viewing list price.
Please call 1-800-766-7000 to place your order or try our site again later.

RASGRP3 Mouse anti-Human, Clone: OTI2C11, Invitrogen™

Mouse Monoclonal Antibody

Manufacturer:  Invitrogen MA526504

Catalog No. PIMA526504



Description

Description

Members of the RAS (see HRAS; MIM 190020) subfamily of GTPases function in signal transduction as GTP/GDP-regulated switches that cycle between inactive GDP- and active GTP-bound states. Guanine nucleotide exchange factors (GEFs), such as RASGRP3, serve as RAS activators by promoting acquisition of GTP to maintain the active GTP-bound state and are the key link between cell surface receptors and RAS activation (Rebhun et al., 2000 [PubMed 10934204]).
Specifications

Specifications

RASGRP3
Monoclonal
1 mg/mL
PBS with 1% BSA, 50% glycerol and 0.02% sodium azide; pH 7.3
Q8IV61
OTTHUMP00000201245; calcium and DAG-regulated guanine nucleotide exchange factor III; guanine nucleotide exchange factor for Rap1; ras guanyl-releasing protein 3
Mouse
IgG2a
100 μL
-20° C, Avoid Freeze/Thaw Cycles
Primary
25780
Immunohistochemistry (Paraffin), Western Blot
OTI2C11
Unconjugated
RASGRP3
Liquid
RASGRP3
Human recombit protein fragment corresponding to amino acids 380-690 of RASGRP3 produced in E.coli
Affinity Chromatography
RUO
Antibody
Monoclonal
Human
Documents
Provide Content Correction

We continue to work to improve your shopping experience and your feedback regarding this content is very important to us. Please use the form below to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Cancel Submit