Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More

RbAp46 Mouse anti-Human, Clone: OTI5A4, Invitrogen™

Mouse Monoclonal Antibody

Manufacturer:  Invitrogen MA525715

Catalog No. PIMA525715


Add to cart

Description

Description

Retinoblastoma protein (Rb) associated protein 46 (RbAp46) is a nuclear protein that has been shown to interact directly with retinoblastoma protein and is a component of the mSin3 corepressor complex as are the histone deacetylase proteins (HDACs). RbAp46 shares about 75% sequence identity with the closely related RbAp48, and appears to be functionally related to the yeast MSI1 protein in that overexpression of RbAp46 suppresses heat shock sensitivity in a RAS pathway dependent manner. RbAp46 has also been demonstrated to be up-regulated by the Wilms ft. tumor suppressor gene product, WT1. Additionally, cells transfected with RbAp46 have shown strong inhibition of growth.
Specifications

Specifications

RbAp46
Monoclonal
0.84 mg/mL
PBS with 1% BSA, 50% glycerol and 0.02% sodium azide; pH 7.3
Q16576
RbAp46, G1/S transition control protein-binding protein RbAp46, RBBP-7, histone acetyltransferase type B subunit 2, histone-binding protein RBBP7, nucleosome-remodeling factor subunit RBAP46, retinoblastoma-binding protein 7, retinoblastoma-binding protein RbAp46, retin
Mouse
IgG1
100 μL
-20° C, Avoid Freeze/Thaw Cycles
Primary
5931
Immunohistochemistry (Paraffin), Western Blot
OTI5A4
Unconjugated
RBBP7
Liquid
RBBP7
Full length human recombit protein of RBBP7 produced in HEK293T cell
Affinity Chromatography
RUO
Antibody
Monoclonal
Human
Documents
Provide Content Correction

We continue to work to improve your shopping experience and your feedback regarding this content is very important to us. Please use the form below to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Cancel Submit