Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More

RGS2 293T Cell Overexpression Lysate (Denatured), Abnova

Denatured GS2 293T cell transient overexpression lysate

Manufacturer:  Abnova Corporation H00005997T01

Catalog No. 89-001-990


Add to cart

Description

Description

Regulator of G protein signaling (RGS) family members are regulatory molecules that act as GTPase activating proteins (GAPs) for G alpha subunits of heterotrimeric G proteins. RGS proteins are able to deactivate G protein subunits of the Gi alpha, Go alpha and Gq alpha subtypes. They drive G proteins into their inactive GDP-bound forms. Regulator of G protein signaling 2 belongs to this family. The protein acts as a mediator of myeloid differentiation and may play a role in leukemogenesis.

  • Transfected cell line: 293T
  • Plasmid: pCMV-RGS2 full-length
  • Host: Human
  • Theoretical MW (kDa): 23.32
  • Storage buffer: 1X sample buffer (50 mM Tris-HCl, 2% SDS, 10% glycerol, 300 mM 2-mercaptoethanol, 0.01% Bromophenol blue)

Western Blotting

Specifications

Specifications

Western Blot
293T
NM_002923
6.8
Transient overexpression cell Lysate was tested with Anti-RGS2 antibody (H00005997-B01) by western blotting
1X Sample Buffer (50 mM Tris-HCl, 2% SDS, 10% glycerol, 300 mM 2-mercaptoethanol, 0.01% Bromophenol blue)
Human
RGS2 293T Cell Transient Overexpression Lysate(Denatured)
24.4
pCMV-RGS2 full-length
100 uL
Store at -80°C. Aliquot to avoid repeated freezing and thawing.
Documents
Provide Content Correction

We continue to work to improve your shopping experience and your feedback regarding this content is very important to us. Please use the form below to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Cancel Submit