Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More

VLDLR Mouse anti-Bovine, Human, Mouse, Rat, Clone: 6A6, Invitrogen™

Mouse Monoclonal Antibody

Manufacturer:  Invitrogen MA524790

Catalog No. PIMA524790


Add to cart

Description

Description

The low density lipoprotein receptor (LDLR) gene family consists of cell surface proteins involved in receptor-mediated endocytosis of specific ligands. This gene encodes a lipoprotein receptor that is a member of the LDLR family and plays important roles in VLDL-triglyceride metabolism and the reelin signaling pathway. Mutations in this gene cause VLDLR-associated cerebellar hypoplasia. Alternative splicing generates multiple transcript variants encoding distinct isoforms for this gene.
Specifications

Specifications

VLDLR
Monoclonal
1 mg/mL
PBS with 0.02% sodium azide; pH 7.4
P98155, P98156, P98166
CARMQ1, CHRMQ1, FLJ35024, very low density lipoprotein receptor, very low-density lipoprotein receptor, VLDL receptor, VLDL-R, VLDLRCH
Mouse
IgG1
100 μL
Store at 4°C short term. For long term storage, store at -20°C, avoiding freeze/thaw cycles.
Primary
22359, 25696, 282123, 7436
Immunocytochemistry, Immunofluorescence, Immunohistochemistry, Immunohistochemistry (Paraffin), Western Blot
6A6
Unconjugated
VLDLR
Liquid
VLDLR
Synthetic peptide corresponding to the C-terminus of human VLDL Receptor.
Protein G
RUO
Antibody
Monoclonal
Bovine, Human, Mouse, Rat
Documents
Provide Content Correction

We continue to work to improve your shopping experience and your feedback regarding this content is very important to us. Please use the form below to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Cancel Submit