Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More
  1. Home
  2. Products
  3. 5000344962

Medchemexpress LLC 5-(4-Hydroxyphenyl)-3H-1,2-dithiole-3-thione | 18274-81-2 | 98.55% | 226.35 | 50 G
SDP

Catalog No. 5000344962
Encompass_Preferred

ADT-OH is a hydrogen sulfide-releasing donor that induces apoptosis and upregulates FADD. It inhibits FAK/Paxillin and shows potential for research in cancer diseases.

  • Induces apoptosis.
  • Upregulates FADD.
  • Inhibits FAK/Paxillin.
  • Significantly inhibits metastasis of TNBC cells.
  • Enhances antitumor activity of celecoxib in human colorectal cancer cells.
  • Promotes self-renewal and antiapoptosis ability of cultured neural precursor cells.
  • Exhibits anti-metastatic activity on malignant melanoma cells.
  • Inhibits human HDAC in HeLa cells.
  • Shows potent anti-metastatic activity in xenograft models.
  • Suppresses tumor metastasis with limited effect on primary tumor growth.

Catalog No. 50-003-44962 Supplier Medchemexpress LLC Supplier No. HY10958250MG
May include imposed supplier surcharges.
Add to Cart
Add to Cart

missing translation for 'validMainframeMsg'

ADT-OH is a hydrogen sulfide-releasing donor that induces apoptosis and upregulates FADD. It inhibits FAK/Paxillin and shows potential for research in cancer diseases.

  • Induces apoptosis.
  • Upregulates FADD.
  • Inhibits FAK/Paxillin.
  • Significantly inhibits metastasis of TNBC cells.
  • Enhances antitumor activity of celecoxib in human colorectal cancer cells.
  • Promotes self-renewal and antiapoptosis ability of cultured neural precursor cells.
  • Exhibits anti-metastatic activity on malignant melanoma cells.
  • Inhibits human HDAC in HeLa cells.
  • Shows potent anti-metastatic activity in xenograft models.
  • Suppresses tumor metastasis with limited effect on primary tumor growth.

Product Title
Select an issue

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.