Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More

Medchemexpress LLC Adenosine | 58-61-7 | 100.0% | 50 G
SDP

Catalog No. 5000405477
Encompass_Preferred

Adenosine is a ubiquitous and BBB-permeable endogenous autacoid, acting through four G protein-coupled receptors (A1, A2A, A2B, and A3). It influences nearly all aspects of cellular physiology, including neuronal activity, vascular function, platelet aggregation, and blood cell regulation. As an extracellular signaling molecule derived from 5'-adenosine triphosphate (ATP) and 5'-adenosine monophosphate (AMP), it plays a critical role in cellular processes.

  • Influences neuronal activity, vascular function, platelet aggregation, and blood cell regulation.
  • Acts via G protein-coupled receptors: A1, A2A, A2B, and A3.
  • Inhibits cell viability and triggers ER stress in HepG2 cells at high concentrations.
  • Induces apoptosis and autophagy in HepG2 cells via the AMPK/mTOR pathway.

Catalog No. 50-004-05477 Supplier Medchemexpress LLC Supplier No. HYB022850G
May include imposed supplier surcharges.
Add to Cart
Add to Cart

missing translation for 'validMainframeMsg'

Adenosine is a ubiquitous and BBB-permeable endogenous autacoid, acting through four G protein-coupled receptors (A1, A2A, A2B, and A3). It influences nearly all aspects of cellular physiology, including neuronal activity, vascular function, platelet aggregation, and blood cell regulation. As an extracellular signaling molecule derived from 5'-adenosine triphosphate (ATP) and 5'-adenosine monophosphate (AMP), it plays a critical role in cellular processes.

  • Influences neuronal activity, vascular function, platelet aggregation, and blood cell regulation.
  • Acts via G protein-coupled receptors: A1, A2A, A2B, and A3.
  • Inhibits cell viability and triggers ER stress in HepG2 cells at high concentrations.
  • Induces apoptosis and autophagy in HepG2 cells via the AMPK/mTOR pathway.

Product Title
Select an issue

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.