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Medchemexpress LLC Eicosapentaenoic Acid | 10417-94-4 | 99.9% | 25 MG
SDP

Catalog No. 5000450782
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Eicosapentaenoic Acid (EPA), also known as Timnodonic acid, is an orally active Omega-3 long-chain polyunsaturated fatty acid (ω-3 LC-PUFA). It acts as a DNA demethylating agent, promoting the re-expression of the tumor suppressor gene CCAAT/enhancer-binding protein δ (C/EBPδ). EPA also activates the RAS/ERK/C/EBPβ pathway through H-Ras intron 1 CpG island demethylation in U937 leukemia cells. Furthermore, Eicosapentaenoic Acid can induce relaxation of vascular smooth muscle cells and vasodilation.

  • Orally active Omega-3 long-chain polyunsaturated fatty acid (ω-3 LC-PUFA).
  • Exhibits DNA demethylating action.
  • Promotes re-expression of the tumor suppressor gene CCAAT/enhancer-binding protein δ (C/EBPδ).
  • Activates RAS/ERK/C/EBPβ pathway via H-Ras intron 1 CpG island demethylation in U937 leukemia cells.
  • Promotes relaxation of vascular smooth muscle cells and vasodilation.
  • Human Endogenous Metabolite.

Catalog No. 50-004-50782 Supplier Medchemexpress LLC Supplier No. 25MGHYB0660R
May include imposed supplier surcharges.
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Eicosapentaenoic Acid (EPA), also known as Timnodonic acid, is an orally active Omega-3 long-chain polyunsaturated fatty acid (ω-3 LC-PUFA). It acts as a DNA demethylating agent, promoting the re-expression of the tumor suppressor gene CCAAT/enhancer-binding protein δ (C/EBPδ). EPA also activates the RAS/ERK/C/EBPβ pathway through H-Ras intron 1 CpG island demethylation in U937 leukemia cells. Furthermore, Eicosapentaenoic Acid can induce relaxation of vascular smooth muscle cells and vasodilation.

  • Orally active Omega-3 long-chain polyunsaturated fatty acid (ω-3 LC-PUFA).
  • Exhibits DNA demethylating action.
  • Promotes re-expression of the tumor suppressor gene CCAAT/enhancer-binding protein δ (C/EBPδ).
  • Activates RAS/ERK/C/EBPβ pathway via H-Ras intron 1 CpG island demethylation in U937 leukemia cells.
  • Promotes relaxation of vascular smooth muscle cells and vasodilation.
  • Human Endogenous Metabolite.

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