Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More
  1. Home
  2. Products
  3. 5000438453

Medchemexpress LLC HZX-02-059 | 2240205-30-3 | 99.4% | 487.48 | 100 MG
SDP

Catalog No. 5000438453
Encompass_Preferred

HZX-02-059 is a potent methuosis inducer and a dual-target PIKfyve/tubulin inhibitor with anticancer activity. It primarily disrupts the balance of endocytosis and exocytosis, leading to the formation of numerous vesicles and inducing cell death. Additionally, HZX-02-059 promotes cell vacuolization, apoptosis, downregulates the p53 pathway, inhibits PI3K/AKT phosphorylation, and suppresses c-Myc and NF-κB transcription.

  • Potent methuosis inducer.
  • Dual-target PIKfyve/tubulin inhibitor.
  • Exhibits anticancer activity.
  • Induces cell vacuolization.
  • Promotes apoptosis.
  • Downregulates the p53 pathway.
  • Inhibits PI3K/AKT phosphorylation.
  • Inhibits c-Myc and NF-κB transcription.

Catalog No. 50-004-38453 Supplier Medchemexpress LLC Supplier No. HY112440100MG
May include imposed supplier surcharges.
Add to Cart
Add to Cart

missing translation for 'validMainframeMsg'

HZX-02-059 is a potent methuosis inducer and a dual-target PIKfyve/tubulin inhibitor with anticancer activity. It primarily disrupts the balance of endocytosis and exocytosis, leading to the formation of numerous vesicles and inducing cell death. Additionally, HZX-02-059 promotes cell vacuolization, apoptosis, downregulates the p53 pathway, inhibits PI3K/AKT phosphorylation, and suppresses c-Myc and NF-κB transcription.

  • Potent methuosis inducer.
  • Dual-target PIKfyve/tubulin inhibitor.
  • Exhibits anticancer activity.
  • Induces cell vacuolization.
  • Promotes apoptosis.
  • Downregulates the p53 pathway.
  • Inhibits PI3K/AKT phosphorylation.
  • Inhibits c-Myc and NF-κB transcription.

Product Title
Select an issue

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.