Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More

Invitrogen™ IBA1 Recombinant Rat Monoclonal Antibody (HL22-RT)

Rat Recombinant Monoclonal Antibody

Supplier:  Invitrogen™ MA538266

Catalog No. PIMA538266


Only null left
Add to Cart

Description

Description

Centrifuge briefly prior to opening.

AIF1 is induced by cytokines and interferon. It is thought to be involved in the negative regulation of growth of vascular smooth muscle cells, which contributes to the anti-inflammatory response to vessel wall trauma.
TRUSTED_SUSTAINABILITY
Specifications

Specifications

IBA1
Recombinant Monoclonal
2 mg/mL
PBS with no preservative
O70200, P55008, P55009
AIF1
Carrier-protein conjugated synthetic peptide encompassing a sequence within the C-terminus region of human Iba1. The exact sequence is proprietary.
100 μL
Primary
Human, Mouse, Rat
Antibody
IgG1
Immunohistochemistry (Frozen), Immunohistochemistry (Paraffin), Western Blot
HL22-RT
Unconjugated
AIF1
AI607846; AIF1; AIF-1; Allograft inflammatory factor 1; balloon angioplasty responsive transcript; balloon angioplasty-responsive transcript 1; Bart1; BART-1; D17H6S50E; DADB-70P7.8; DASS-82G15.2; Em:AF129756.17; G1; Iba; Iba1; interferon gamma responsive transcript; ionized calcium binding adapter molecule 1; ionized calcium-binding adapter molecule 1; IRT1; IRT-1; microglia response factor; Mrf1; MRF-1; protein BART-1; protein G1; testis specific
Rat
Protein G
RUO
11629, 199, 29427
-20°C, Avoid Freeze/Thaw Cycles
Liquid
Product Suggestions

Product Suggestions

Videos
SDS
Documents

Documents

Product Certifications
Promotions

Promotions

Product Content Correction

Your input is important to us. Please complete this form to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Your feedback has been submitted: Thank you for helping us improve our website.