Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More

Invitrogen™ METTL3 Monoclonal Antibody (OTI1B7)

Mouse Monoclonal Antibody

Supplier:  Invitrogen™ MA527527

Catalog No. PIMA527527


Only null left
Add to Cart

Description

Description

METTL3 Monoclonal Antibody for Western Blot

METTL3 is the 70 kDa subunit of MT-A which is part of N6-adenosine-methyltransferase. This enzyme is involved in the posttranscriptional methylation of internal adenosine residues in eukaryotic mRNAs, forming N6-methyladenosine.
TRUSTED_SUSTAINABILITY
Specifications

Specifications

METTL3
Monoclonal
1 mg/mL
PBS with 1% BSA, 50% glycerol and 0.02% sodium azide; pH 7.3
Q86U44, Q8C3P7
METTL3
Human recombinant protein fragment corresponding to amino acids 1-284 of METTL3 produced in E.coli.
100 μL
Primary
Human, Mouse, Rat
Antibody
IgG1
Western Blot
OTI1B7
Unconjugated
METTL3
2310024F18Rik; adoMet-binding subunit of the human mRNA (N6-adenosine)-methyltransferase; fj82g07; hMETTL3; IME4; M6A; m6a methyltransferase; methyltransferase like 3; methyltransferase-like 3; methyltransferase-like protein 3; METTL3; mRNA (2'-O-methyladenosine-N(6)-)-methyltransferase; mRNA m(6)A methyltransferase; MTA70; MT-A70; N6-adenosine-methyltransferase 70 kDa subunit; N6-adenosine-methyltransferase catalytic subunit; N6-adenosine-methyltransferase subunit METTL3; Spo8; Unknown (protein for MGC:77093); wu:fj82g07; zgc:77093
Mouse
Affinity Chromatography
RUO
361035, 56335, 56339
-20°C, Avoid Freeze/Thaw Cycles
Liquid
Product Suggestions

Product Suggestions

Videos
SDS
Documents

Documents

Product Certifications
Promotions

Promotions

Product Content Correction

Your input is important to us. Please complete this form to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Your feedback has been submitted: Thank you for helping us improve our website.