Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More

Invitrogen™ MyD88 Polyclonal Antibody

Goat Polyclonal Antibody

Supplier:  Invitrogen™ PA19072

Catalog No. 01-674-656


Only null left
Add to Cart

Description

Description

This antibody is predicted to react with canine, mouse and rat based on sequence homology. This antibody is tested in Peptide ELISA: antibody detection limit dilution 128,000.

Myeloid differentiation Marker 88 (MyD88) is an adaptor molecule for the Toll/IL-1R and is involved in the inflammatory response induced by IL-1, IL-18 and LPS. MyD88 recruits IRAK to IL-1 receptor complex in response to IL-1. This pathway further leads to activation of NF-kB. MyD88 has a wide tissue distribution.
TRUSTED_SUSTAINABILITY
Specifications

Specifications

MyD88
Polyclonal
Unconjugated
MYD88
mutant myeloid differentiation primary response 88; Myd88; MYD88D; myeloid differentiation primary response 88; myeloid differentiation primary response gene (88); myeloid differentiation primary response gene 88; myeloid differentiation primary response protein MyD88
Goat
Ammonium Sulfate Precipitation
RUO
4615
-20°C, Avoid Freeze/Thaw Cycles
Liquid
Flow Cytometry, Immunohistochemistry (Paraffin), Western Blot, Immunocytochemistry
0.5 mg/mL
TBS with 0.5% BSA and 0.02% sodium azide; pH 7.3
Q99836
MYD88
Internal sequence corresponding to amino acids C-IKYKAMKKEFP (aa 255-266).
100 μg
Primary
Human
Antibody
IgG
Product Suggestions

Product Suggestions

Videos
SDS
Documents

Documents

Product Certifications
Promotions

Promotions

Product Content Correction

Your input is important to us. Please complete this form to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Your feedback has been submitted: Thank you for helping us improve our website.