Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More
Our pricing system is unavailable. You are viewing list price.
Please call 1-800-766-7000 to place your order or try our site again later.

NQO2 Mouse anti-Canine, Human, Non-human primate, Clone: OTI1G2, Invitrogen™

Mouse Monoclonal Antibody

Manufacturer:  Invitrogen MA525981

Catalog No. PIMA525981



Description

Description

NQO2 (EC 1.10.99.2) is a flavoprotein that catalyzes the 2-electron reduction of various quinones, redox dyes, and the vitamin K menadione. NQO2 predomitly uses dihydronicotinamide riboside (NRH) as the electron donor (summary by Wu et al., 1997 [PubMed 9367528]).
Specifications

Specifications

NQO2
Monoclonal
1 mg/mL
PBS with 1% BSA, 50% glycerol and 0.02% sodium azide; pH 7.3
P16083
DHQV;DIA6;EC 1.10.99.2;MGC94180;NAD(P)H dehydrogenase quinone 2;NAD(P)H menadione oxidoreductase 1 dioxin inducible 2;NAD(P)H menadione oxidoreductase 2 dioxin inducible;NMOR2;NQO 2;NQO2;NQO2_HUMAN;NRH dehydrogenase [quinone] 2;NRH dehydrogenase;NRH:quinone oxidoreductase 2;OTTHUMP00000015948;OTTHUMP00000015949;OTTHUMP00000015953;Ox 2;Ox2;QR2;Quinone;Quinone reductase 2;Ribosyldihydronicotinamide dehydrogenase [quinone];Ribosyldihydronicotinamide dehydrogenase;
Mouse
IgG1
100 μL
-20° C, Avoid Freeze/Thaw Cycles
Primary
4835, 606932, 707675
Immunocytochemistry, Immunofluorescence, Immunohistochemistry (Paraffin), Western Blot
OTI1G2
Unconjugated
NQO2
Liquid
NQO2
Full length human recombit protein of NQO2 produced in HEK293T cell
Affinity Chromatography
RUO
Antibody
Monoclonal
Canine, Human, Non-human primate
Documents
Provide Content Correction

We continue to work to improve your shopping experience and your feedback regarding this content is very important to us. Please use the form below to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Cancel Submit