Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More

Invitrogen™ PR3 Monoclonal Antibody (PR3G-2), FITC

Mouse Monoclonal Antibody

Supplier:  Invitrogen™ MA549093

Catalog No. FERMA549093


Only null left
Add to Cart

Description

Description

The monoclonal antibody PR3G-2 was produced by immunization of mice with a crude granule extract.

Serine protease that degrades elastin, fibronectin, laminin, vitronectin, and collagen types I, III, and IV (in vitro) (PubMed:3198760, PubMed:2033050, PubMed:28240246). By cleaving and activating receptor F2RL1/PAR-2, enhances endothelial cell barrier function and thus vascular integrity during neutrophil transendothelial migration (PubMed:23202369). May play a role in neutrophil transendothelial migration, probably when associated with CD177 (PubMed:22266279). [UniProt].
TRUSTED_SUSTAINABILITY
Specifications

Specifications

PR3
Monoclonal
100 μg/mL
PBS with 1% BSA and 0.02% sodium azide
P24158
PRTN3
Monoclonal antibody PR3G-2 reacts with human proteinase 3 (PR3), a 30 kDa protein.
100 μg
Primary
Human
Antibody
IgG1
Flow Cytometry, Functional Assay, Inhibition Assays, Western Blot
PR3G-2
FITC
PRTN3
ACPA; AGP7; azurophil granule protein 7; CANCA; C-ANCA; C-ANCA antigen; leukocyte proteinase 3; MBN; MBT; mPR3; Myeloblastin; neutrophil proteinase 4; NP4; NP-4; P29; PR3; PR-3; Proteinase 3; PRTN3; serine proteinase, neutrophil; wegener autoantigen; Wegener granulomatosis autoantigen
Mouse
Protein G
RUO
5657
4°C, store in dark
Liquid
Product Suggestions

Product Suggestions

Videos
SDS
Documents

Documents

Product Certifications
Promotions

Promotions

Product Content Correction

Your input is important to us. Please complete this form to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Your feedback has been submitted: Thank you for helping us improve our website.