Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More

Invitrogen™ Adalimumab Recombinant Human Monoclonal Antibody
GREENER_CHOICE

Human Recombinant Monoclonal Antibody

Supplier:  Invitrogen™ MA541785

Catalog No. PIMA541785


Only null left
Add to Cart
This item is not returnable. View return policy

Description

Description

Endotoxin level < 0.01EU/μg by LAL method Use a manual defrost freezer and avoid repeated freeze thaw cycles. Store at 2 to 8 °C for one week. Store at -20 to -80 °C for twelve months from the date of receipt.

Adalimumab (Humira) is a humanized monoclonal antibody that is approved by the U.S. Food and Drug Administration for the treatment of patients with rheumatoid arthritis, psoriatic arthritis, ankylosing spondylitis, Crohn's disease, ulcerative colitis, chronic psoriasis, hidradenitis suppurativa, and juvenile idiopathic arthritis. Adalimumab binds to the TNF alpha (tumor necrosis factor-alpha), and blocks its interaction with TNF alpha receptors, reducing the inflammatory response of autoimmune diseases.
TRUSTED_SUSTAINABILITY
Specifications

Specifications

Adalimumab
Recombinant Monoclonal
Unconjugated
D2E7; Humira
Human TNFSF2/TNF-alpha/TNFA.
100 μg
Primary
-20°C, Avoid Freeze/Thaw Cycles
Liquid
Chemical
ELISA, Flow Cytometry
1.9 mg/mL
PBS with no preservative; pH 7.4
Human
Protein A/G
RUO
Human
Antibody
IgG1 κ
Product Suggestions

Product Suggestions

Videos
SDS
Documents

Documents

Product Certifications
Promotions

Promotions

Product Content Correction

Your input is important to us. Please complete this form to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Your feedback has been submitted: Thank you for helping us improve our website.