Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More

AMOT Rabbit anti-Human, Mouse, Rat, Polyclonal, Proteintech
SDP

Rabbit Polyclonal Antibody

Manufacturer:  Proteintech Group Inc 168701AP150UL

Encompass_Preferred

 View more versions of this product

Catalog No. 50-173-1230


Add to cart

Description

Description

This antibody recognizes p130 only.

This gene belongs to the motin family of angiostatin binding proteins characterized by conserved coiled-coil domains and C-terminal PDZ binding motifs. The encoded protein is expressed predomitly in endothelial cells of capillaries as well as larger vessels of the placenta where it may mediate the inhibitory effect of angiostatin on tube formation and the migration of endothelial cells toward growth factors during the formation of new blood vessels. Alternative splicing results in multiple transcript variants encoding different isoforms.
Specifications

Specifications

AMOT
Polyclonal
Unconjugated
AMOT
AMOT, angiomotin, KIAA1071
Rabbit
Antigen Affinity Chromatography
RUO
154796, 27494, 300289
-20°C
Liquid
Western Blot
0.34 mg/mL
PBS with 50% glycerol and 0.02% sodium azide; pH 7.3
Q4VCS5, Q8VHG2
AMOT
Peptide
150 μL
Primary
Human, Mouse, Rat
Antibody
IgG
SDS
Documents
Product Certifications
Promotions

Promotions

Provide Content Correction

The Fisher Scientific Encompass Program offers items which are not part of our distribution portfolio. These products typically do not have pictures or detailed descriptions. However, we are committed to improving your shopping experience. Please use the form below to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Cancel Submit