Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More

Invitrogen™ ANO7 Polyclonal Antibody

Rabbit Polyclonal Antibody

Supplier:  Invitrogen™ PA5104532

Catalog No. PIPA5104532


Only null left
Add to Cart

Description

Description

Antibody detects endogenous levels of total TM16G.

Has calcium-dependent phospholipid scramblase activity; scrambles phosphatidylserine, phosphatidylcholine and galactosylceramide. Does not exhibit calcium-activated chloride channel (CaCC) activity. May play a role in cell-cell interactions.
TRUSTED_SUSTAINABILITY
Specifications

Specifications

ANO7
Polyclonal
Unconjugated
ANO7
ANO7; anoctamin 7; anoctamin-7; Dresden transmembrane protein of the prostate; Dresden-transmembrane protein of the prostate; DTMPP; D-TMPP; IPCA5; IPCA-5; New gene expressed in prostate; NGEP; PCANAP5; PCANAP5L; prostate cancer associated protein 5; Prostate cancer-associated protein 5; TMEM16G; Transmembrane protein 16G
Rabbit
Affinity chromatography
RUO
50636
-20°C
Liquid
Immunohistochemistry (Paraffin), Western Blot, Immunocytochemistry
1 mg/mL
PBS with 50% glycerol and 0.02% sodium azide; pH 7.4
Q6IWH7
ANO7
A synthesized peptide derived from human ANO7(Accession Q6IWH7), corresponding to amino acid residues Y457-M507.
100 μL
Primary
Human
Antibody
IgG
Product Suggestions

Product Suggestions

Videos
Safety and Handling

Safety and Handling

WARNING: Cancer - www.P65Warnings.ca.gov
SDS
Documents

Documents

Product Certifications
Promotions

Promotions

Product Content Correction

Your input is important to us. Please complete this form to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Your feedback has been submitted: Thank you for helping us improve our website.