Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More

BIK, Rabbit, Monoclonal Antibody, Abnova™

Rabbit monoclonal antibody raised against a human BIK peptide using ARM Technology.

Supplier:  Abnova Corporation H00000638K

Catalog No. 89-205-386


Includes: Up to three rabbit IgG clones of 1mg each will be delivered to customer.

Description

Description

The protein encoded by this gene is known to interact with cellular and viral survival-promoting proteins, such as BCL2 and the Epstein-Barr virus in order to enhance programed cell death. Because its activity is suppressed in the presence of survival-promoting proteins, this protein is suggested as a likely target for antiapoptotic proteins. This protein shares a critical BH3 domain with other death-promoting proteins, BAX and BAK. [provided by RefSeq

Library Construction: Nonfusion antibody library from rabbit spleen (ARM Technology)
Expression: Overexpression vector and transfection into 293H cell line

Order Info

  1. Customer may provide cell or tissue lysate for antibody screening.
  2. Rabbit monoclonal antibody generated by ARM technology is amenable to antibody engineering including F(ab)2, IgG, scFv and different Fc and non-Fc conjugates per customer request.

Specifications

Specifications

BIK
Monoclonal
Unconjugated
In 1X PBS, pH 7.4
BIK
BIK
A synthetic peptide of human BIK
RUO
638
Store at -20°C or lower. Aliquot to avoid repeated freezing and thawing.
IgG
ELISA, Western Blot
1mg
BCL2-interacting killer (apoptosis-inducing)
BIK
BIP1, BP4, NBK
Rabbit
1 mg x up to 3 Abs
Primary
Human
Liquid
Product Suggestions

Product Suggestions

Videos
SDS
Documents

Documents

Product Certifications
Promotions

Promotions

Product Content Correction

Your input is important to us. Please complete this form to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Your feedback has been submitted: Thank you for helping us improve our website.