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CDw93 (C1qRp) Mouse anti-Human, NA/LE, Unlabeled, Clone: R139, BD

Catalog No. BDB552954 Shop All BD Biosciences Products
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BDB552954 0.5 mg
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Catalog No. BDB552954 Supplier BD Biosciences Supplier No. 552954
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Mouse Monoclonal Antibody

The immunogen used to raise R139 antibody was C1q-binding protein preparation derived from U937 cell lysates, as described. Human CDw93 (C1qRp) is a 631 AA, single chain type I membrane glycoprotein expressed on cells of myeloid origin, endothelial cells, and hematopoietic progenitor cells. Human, murine and rat protein sequences have been deduced from cDNA clones and are known to be similar in sequence and organization. CDw93 (C1qRp) binds C1q, the recognition subunit of the first component (C1) of the complement pathway, as well as MBL (Mannose-binding-lectin) and SPA (Pulmonary Surfactant Protein A). Multivalent interaction of CDw93 (C1qRp) expressing cells with C1q, MBL, and SPA, induces enhancement of phagocytosis of suboptimally opsonized particles and/or cellular debris. Antibody R139 neutralizes/blocks C1q-mediated enhancement of phagocytosis, as reported. In addition clone R139 is suitable to detect CDw93 (C1qRp) expression on cells by flow cytometry, CDw93 (C1qRp) in cellular lysates by Western blotting or immunoprecipitation. CDw93 (C1qRp) has been reported to define a human stem cell population with hematopoietic and hepatic potential.

TRUSTED_SUSTAINABILITY

Specifications

Antigen CDw93 (C1qRp)
Applications Flow Cytometry
Classification Monoclonal
Clone R139
Concentration 1.0mg/mL
Conjugate Unconjugated
Description C1qRp
Formulation No azide/low endotoxin: Aqueous buffered solution containing no preservative, 0.2μm sterile filtered. Endotoxin level is ≤0.01 EU/μg (≤0.001 ng/μg) of protein as determined by the LAL assay.
Host Species Mouse
Purification Method Affinity Purified
Quantity 0.5 mg
Regulatory Status RUO
Research Discipline Cell Biology
Primary or Secondary Primary
Target Species Human
Content And Storage Store undiluted at 4°C
Isotype IgG2b κ
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For Research Use Only.

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