Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More

anti-CXCR2/IL-8 RB, Alexa Fluor 700, Clone: 48311, R&D Systems™

Supplier:  R&D Systems FAB331N025

 View more versions of this product

Catalog No. FAB331N025

This item is not returnable. View return policy

Description

Description

Host Species: Mouse
Clone: 48311
Isotype: IgG2A
Species Reactivity: Hu
Immunogen: NS0 mouse myeloma cell line transfected with human CXCR2/IL-8 RB. Met1-Leu355

Applications:

Flow Cytometry

Specifications

Specifications

CXCR2/IL-8 RB
Monoclonal
Alexa Fluor 700
AAB25880
CXCR2
NS0 mouse myeloma cell line transfected with human CXCR2/IL-8 RB, Met1-Leu355, Accession # AAB25880
25 Tests
3579
Human
Purified
Flow Cytometry
48311
Flow Cytometry 5 uL/10^6 cells
beta, CD182, CD182 antigen, CDw128b, chemokine (C-X-C motif) receptor 2, chemokine (CXC) receptor 2, C-X-C chemokine receptor type 2, CXC-R2, CXCR-2, CXCR2 gene for IL8 receptor type B, GRO/MGSA receptor, High affinity interleukin-8 receptor B, IL-8 receptor type 2, IL-8R B, IL8R2, IL8RA, IL8RB, IL-8RB, interleukin 8 receptor B, interleukin 8 receptor type 2, interleukin-8 receptor type B
Mouse
Protein A or G purified from ascites
Primary
Detects human CXCR2/IL-8 RB transfected NS0 cells but not the parental cell line. It does not cross-react with CXCR1 (IL-8 RA).
Protect from light. Do not freeze. 12 months from date of receipt, 2 to 8 °C as supplied.
IgG2a
Product Suggestions

Product Suggestions

Videos
SDS
Documents

Documents

Product Certifications
Promotions

Promotions

Product Content Correction

Your input is important to us. Please complete this form to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Your feedback has been submitted: Thank you for helping us improve our website.