Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More

SLC27A5 Mouse anti-Human, Clone: 390917, Invitrogen™

Mouse Monoclonal Antibody

Manufacturer:  Invitrogen MA524125

Catalog No. PIMA524125


Add to cart

Description

Description

This antibody stains human FATP5 transfectants but not irrelevant transfectants. Reconstitute at 0.5 mg/mL in sterile PBS. Endoxin level is <0.10 EU per 1 µg of the antibody by the LAL method.

The protein encoded by this gene is an isozyme of very long-chain acyl-CoA synthetase (VLCS). It is capable of activating very long-chain fatty-acids containing 24- and 26-carbons. It is expressed in liver and associated with endoplasmic reticulum but not with peroxisomes. Its primary role is in fatty acid elongation or complex lipid synthesis rather than in degradation. This gene has a mouse ortholog.
Specifications

Specifications

SLC27A5
Monoclonal
0.5 mg/mL
PBS with 5% trehalose and No Preservative
Q9Y2P5
ACSB, ACSVL6, BACS, BAL, FACVL3, FATP-5, FATP5, VLACSR, VLCS-H2, VLCSH2, BA-CoA ligase, VLACS-related, bile acid-CoA ligase, bile acyl-CoA synthetase, cholate--CoA ligase, fatty acid transport protein 5, fatty-acid-Coenzyme A ligase, very long-chain 3, solute carrier family 27 member 5, very long-chain acyl-CoA synthetase homolog 2, ve
Mouse
IgG2A
100 μg
-20° C, Avoid Freeze/Thaw Cycles
Primary
10998
Flow Cytometry
390917
Unconjugated
SLC27A5
Lyophilized
SLC27A5
HEK293 human embryonic kidney cell line transfected with human FATP5 Met1-Leu690
Protein A/G
RUO
Antibody
Monoclonal
Human
Documents
Provide Content Correction

We continue to work to improve your shopping experience and your feedback regarding this content is very important to us. Please use the form below to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Cancel Submit