Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More

Invitrogen™ JDP2 Polyclonal Antibody

Rabbit Polyclonal Antibody

Supplier:  Invitrogen™ PA550994

Catalog No. PA550994


Only null left
Add to Cart

Description

Description

JDP2 Polyclonal Antibody detects endogenous levels of JDP2 protein.

Component of the AP-1 transcription factor that represses transactivation mediated by the Jun family of proteins. Involved in a variety of transcriptional responses associated with AP-1 such as UV-induced apoptosis, cell differentiation, tumorigenesis and antitumogeneris. Can also function as a repressor by recruiting histone deacetylase 3/HDAC3 to the promoter region of JUN. May control transcription via direct regulation of the modification of histones and the assembly of chromatin.
TRUSTED_SUSTAINABILITY
Specifications

Specifications

JDP2
Polyclonal
Unconjugated
JDP2
c-Jun dimerization protein 2; Jdp2; Jdp-2; Jun dimerization protein 2; Jundm2; Jundp2; progesterone receptor co-activator; TIF
Rabbit
Antigen affinity chromatography
RUO
116674, 122953, 81703
-20°C, Avoid Freeze/Thaw Cycles
Liquid
ELISA, Western Blot
1 mg/mL
PBS with 0.5% BSA, 50% glycerol and 0.02% sodium azide; pH 7.4
P97875, Q78E65, Q8WYK2
JDP2
Synthesized peptide derived from human JDP2 around the non-phosphorylation site of T148.
100 μL
Primary
Human, Mouse, Rat
Antibody
IgG
Product Suggestions

Product Suggestions

Videos
SDS
Documents

Documents

Product Certifications
Promotions

Promotions

Product Content Correction

Your input is important to us. Please complete this form to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Your feedback has been submitted: Thank you for helping us improve our website.