Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More

KLRB1 Mouse anti-Human, Clone:4C6A11, Abnova™

Mouse monoclonal antibody raised against partial recombinant human KLRB1.

Supplier:  Abnova Corporation MAB17997

Catalog No. 89-334-329


Only null left
Add to Cart

Description

Description

Natural killer (NK) cells are lymphocytes that mediate cytotoxicity and secrete cytokines after immune stimulation. Several genes of the C-type lectin superfamily, including the rodent NKRP1 family of glycoproteins, are expressed by NK cells and may be involved in the regulation of NK cell function. The KLRB1 protein contains an extracellular domain with several motifs characteristic of C-type lectins, a transmembrane domain, and a cytoplasmic domain. The KLRB1 protein is classified as a type II membrane protein because it has an external C terminus. [provided by RefSeq]
Specifications

Specifications

KLRB1
Monoclonal
Unconjugated
ELISA (1:10000)
Flow Cytometry (1:200-1:400)
Western Blot (1:100-1:500)
The optimal working dilution should be determined by the end user.
KLRB1
Mouse
100 μg
Human
Antibody
IgG2b
ELISA, Flow Cytometry, Western Blot
4C6A11
killer cell lectin-like receptor subfamily B, member 1
In PBS (0.05% sodium azide)
CD161, CLEC5B, MGC138614, NKR, NKR-P1, NKR-P1A, NKRP1A, hNKR-P1A
Recombinant protein corresponding to amino acids 67-225 of human KLRB1.
3820
Store at 4°C. For long term storage store at -20°C.
Aliquot to avoid repeated freezing and thawing.
Liquid
Product Suggestions

Product Suggestions

Videos
SDS
Documents

Documents

Product Certifications
Promotions

Promotions

Product Content Correction

Your input is important to us. Please complete this form to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Your feedback has been submitted: Thank you for helping us improve our website.