Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More

Villin Ab-1 Mouse Monoclonal Antibody, Epredia™

Villin Ab-1, Mouse Monoclonal Ab is one marker in a comprehensive antibody portfolio that addresses all major disease states, ensuring the ideal tests are always available.

Manufacturer:  Epredia MS1499RQ

 View more versions of this product

Catalog No. MS1499RQ

This item has been discontinued by the manufacturer and is no longer available. Please call customer service for assistance: 1-800-766-7000.



Villin can cap, nucleate, sever and bundle actin in a Ca and phosphoinositide-regulated manner. It is associated with the microvillar actin core bundle of intestinal and renal brush border implicated in adsorption. Villin is composed of six repeats, each containing 150 residues that together constitute the core domain followed by the carboxyl-terminal headpiece domain of 87 residues. The core domain retains the Ca dependent capping nucleating and severing activity, whereas the headpiece domain contributes towards actin filament bundling and binding F-actin, independently of Ca

Host Species: Mouse

Clone: CWWB1

Isotype: IgG1

Species Reactivity: Human. Others not known.

Immunogen: Human Villin protein

Molecular Weight: 92.5kDa

Positive Control: Small intestine and kidney

Cellular Localization: Cytoplasmic

Recommended for:

  • Immunohistochemistry (Formalin/paraffin)


Villin Ab-1
Human Villin protein
Immunohistochemistry (Paraffin)
12 mL
Cytoskeletal Protein
Ready to Use
Product Certifications


Provide Content Correction

We continue to work to improve your shopping experience and your feedback regarding this content is very important to us. Please use the form below to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Cancel Submit