Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More

OLIG2, Mouse, Clone: 1G11, Abnova™

Mouse monoclonal antibody raised against recombinant OLIG2.

Supplier:  Abnova Corporation MAB10287

Catalog No. 89-108-474


Only null left
Add to Cart

Description

Description

This gene encodes a basic helix-loop-helix transcription factor which is expressed in oligodendroglial tumors of the brain. The protein is an essential regulator of ventral neuroectodermal progenitor cell fate. The gene is involved in a chromosomal translocation t(14;21)(q11.2;q22) associated with T-cell acute lymphoblastic leukemia. Its chromosomal location is within a region of chromosome 21 which has been suggested to play a role in learning deficits associated with Down syndrome. [provided by RefSeq

Specifications

Specifications

OLIG2
Monoclonal
Unconjugated
ELISA (1:10000) Western Blot (1:500-1:2000) Immunohistochemistry (1:200-1:1000) Immunofluorescence (1:200-1:1000) The optimal working dilution should be determined by the end user.
OLIG2
OLIG2
Recombinant protein corresponding to human OLIG2.
RUO
10215
Store at 4°C. For long term storage store at -20°C.
Aliquot to avoid repeated freezing and thawing.
IgG1
ELISA, Immunofluorescence, Immunohistochemistry, Western Blot
1G11
Mouse monoclonal antibody raised against recombinant OLIG2.
In ascites (0.03% sodium azide)
BHLHB1/OLIGO2/PRKCBP2/RACK17/bHLHe19
Mouse
100 μL
Primary
Human
Liquid
Product Suggestions

Product Suggestions

Videos
SDS
Documents

Documents

Product Certifications
Promotions

Promotions

Product Content Correction

Your input is important to us. Please complete this form to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Your feedback has been submitted: Thank you for helping us improve our website.