Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More

RXRG, Mouse, Clone: 1B2, Abnova™

Mouse monoclonal antibody raised against a partial recombinant RXRG.

Supplier:  Abnova Corporation H00006258M02A

Catalog No. 89-105-382


Only null left
Add to Cart

Description

Description

This gene encodes a member of the retinoid X receptor (RXR) family of nuclear receptors which are involved in mediating the antiproliferative effects of retinoic acid (RA). This receptor forms dimers with the retinoic acid, thyroid hormone, and vitamin D receptors, increasing both DNA binding and transcriptional function on their respective response elements. This gene is expressed at significantly lower levels in nonsmall cell lung cancer cells. Alternate transcriptional splice variants, encoding different isoforms, have been characterized. [provided by RefSeq

Sequence: MYGNYSHFMKFPAGYGGCSSPALQLLLSTSLG
Specifications

Specifications

RXRG
Monoclonal
Unconjugated
ascites with no preservative
NM_001009598
RXRG
RXRG (-,1 a.a. ∽ 33 a.a) partial recombinant protein with GST tag. MW of the GST tag alone is 26 KDa.
RUO
6258
Store at -20°C or lower. Aliquot to avoid repeated freezing and thawing.
Ascites
ELISA
1B2
Mouse monoclonal antibody raised against a partial recombinant RXRG.
RXRG
NR2B3/RXRC
Mouse
200 μL
Primary
Human
Antibody
IgM κ
Product Suggestions

Product Suggestions

Videos
SDS
Documents

Documents

Product Certifications
Promotions

Promotions

Product Content Correction

Your input is important to us. Please complete this form to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Your feedback has been submitted: Thank you for helping us improve our website.