Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More

Invitrogen™ NIM1 Polyclonal Antibody

Rabbit Polyclonal Antibody

Supplier:  Invitrogen™ PA5113103

Catalog No. PIPA5113103


Only null left
Add to Cart

Description

Description

NIM1 Polyclonal Antibody for Western Blot, IHC (P), ELISA

NIM1K serine/threonine kinase or NIM1 belongs to CAMK kinase family. Unlike to other AMPK-related kinases, NIM1 cannot be phosphorylated and activated by LKB1. Two somatic mutations (P333S and P411T) were found in lung neuroendocrine carcinoma and lung large cell carcinoma samples. NIM1 gene ontology (GO) annotations include cellular response to glucose starvation; intracellular signal transduction; negative regulation of TOR signaling; protein phosphorylation.
TRUSTED_SUSTAINABILITY
Specifications

Specifications

NIM1
Polyclonal
Unconjugated
NIM1K
E130304F04Rik; Nim1; NIM1 serine/threonine protein kinase; NIM1 serine/threonine-protein kinase; NIM1K; Serine/threonine-protein kinase NIM1
Rabbit
Protein G
RUO
167359
-20°C or -80°C if preferred
Liquid
ELISA, Immunohistochemistry (Paraffin), Western Blot
5 mg/mL
PBS with 50% glycerol and 0.03% ProClin 300; pH 7.4
Q8IY84
NIM1K
Recombinant Human Serine/threonine-protein kinase NIM1 protein (1-436AA).
100 μg
Primary
Human
Antibody
IgG
Product Suggestions

Product Suggestions

Videos
Safety and Handling

Safety and Handling

WARNING: Cancer - www.P65Warnings.ca.gov
SDS
Documents

Documents

Product Certifications
Promotions

Promotions

Product Content Correction

Your input is important to us. Please complete this form to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Your feedback has been submitted: Thank you for helping us improve our website.