Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More

Invitrogen™ PBXIP1 Polyclonal Antibody

Rabbit Polyclonal Antibody

Supplier:  Invitrogen™ PA5106307

Catalog No. PIPA5106307


Only null left
Add to Cart

Description

Description

Antibody detects endogenous levels of total PBXIP1.

Regulator of pre-B-cell leukemia transcription factors (BPXs) function. Inhibits the binding of PBX1-HOX complex to DNA and blocks the transcriptional activity of E2A-PBX1. Tethers estrogen receptor-alpha (ESR1) to microtubules and allows them to influence estrogen receptors-alpha signaling.
TRUSTED_SUSTAINABILITY
Specifications

Specifications

PBXIP1
Polyclonal
Unconjugated
PBXIP1
4732463H20Rik; hematopoietic PBX-interacting protein; HPIP; PBX homeobox interacting protein 1; PBXIP1; pre B cell leukemia transcription factor interacting protein 1; pre-B-cell leukemia homeobox interacting protein 1; pre-B-cell leukemia transcription factor interacting protein 1; pre-B-cell leukemia transcription factor-interacting protein 1
Rabbit
Affinity chromatography
RUO
229534, 310644, 57326
-20°C
Liquid
Immunohistochemistry (Paraffin), Western Blot
1 mg/mL
PBS with 50% glycerol and 0.02% sodium azide; pH 7.4
A2VD12, Q3TVI8, Q96AQ6
PBXIP1
A synthesized peptide derived from human PBXIP1(Accession Q96AQ6), corresponding to amino acid residues G630-E680.
100 μL
Primary
Human, Mouse, Rat
Antibody
IgG
Product Suggestions

Product Suggestions

Videos
Safety and Handling

Safety and Handling

WARNING: Cancer - www.P65Warnings.ca.gov
SDS
Documents

Documents

Product Certifications
Promotions

Promotions

Product Content Correction

Your input is important to us. Please complete this form to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Your feedback has been submitted: Thank you for helping us improve our website.