Promotional price valid on web orders only. Your contract pricing may differ. Interested in signing up for a dedicated account number?
Learn More

STK39 (phospho S325), Rabbit anti-Human, Mouse, Rat, Polyclonal Antibody, Abnova™

Rabbit polyclonal antibody raised against synthetic phosphopeptide of human STK39.

Supplier:  Abnova Corporation PAB29219

Catalog No. 89-130-653


Only null left
Add to Cart

Description

Description

This gene encodes a serine/threonine kinase that is thought to function in the cellular stress response pathway. The kinase is activated in response to hypotonic stress, leading to phosphorylation of several cation-chloride-coupled cotransporters. The catalytically active kinase specifically activates the p38 MAP kinase pathway, and its interaction with p38 decreases upon cellular stress, suggesting that this kinase may serve as an intermediate in the response to cellular stress. [provided by RefSeq

Specifications

Specifications

STK39
Polyclonal
Rabbit polyclonal antibody raised against synthetic phosphopeptide of human STK39.
In PBS, pH 7.4 (without Mg2+ and Ca2+), (50% glycerol, 0.02% sodium azide)
DCHT/DKFZp686K05124/PASK/SPAK
Rabbit
Affinity purification
RUO
27347
Human, Mouse, Rat
Liquid
Immunohistochemistry (PFA fixed)
Unconjugated
Immunohistochemistry (1:50-1:100) The optimal working dilution should be determined by the end user.
STK39
STK39
Synthetic phosphopeptide (conjugated with KLH) corresponding to residues surrounding S325 of human STK39.
100 μL
Primary
This antibody detects endogenous levels of STK39 only when phosphorylated at serine 325.
Store at -20°C.
Avoid repeated freezing and thawing.
Product Suggestions

Product Suggestions

Videos
SDS
Documents

Documents

Product Certifications
Promotions

Promotions

Product Content Correction

Your input is important to us. Please complete this form to provide feedback related to the content on this product.

Product Title

By clicking Submit, you acknowledge that you may be contacted by Fisher Scientific in regards to the feedback you have provided in this form. We will not share your information for any other purposes. All contact information provided shall also be maintained in accordance with our Privacy Policy.

Your feedback has been submitted: Thank you for helping us improve our website.

For Research Use Only